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Environmental and occupational health

Public Health (BPH / B.Sc Public Health) · Programmes and populations · free study notes for exams, placements and interviews

In short

Environmental health is the branch of public health that deals with the physical, chemical and biological agents in air, water, soil, housing and climate that affect human health; occupational health deals with the same agents plus ergonomic and psychosocial hazards as they arise from work. In India they carry an enormous disease burden — ambient and household air pollution, unsafe water and sanitation, and occupational dust together account for a large share of deaths and DALYs, with air pollution alone linked to over a million deaths a year in Indian burden-of-disease estimates. The subject is examined in BPH/B.Sc Public Health papers on environmental health, occupational health and epidemiology of non-communicable disease, and in competitive exams (UPSC, state health services, NIHFW/AIIMS MPH entrance). It is used daily by public health officers, EHS and HSE executives in factories, municipal health departments, ESIC and state factory inspectorates, NGOs and ESG/sustainability teams.

Explained

Concepts, scope and the exposure–disease pathway

Air: ambient, household and Indian standards

Water, sanitation, waste and vectors

Occupational hazards, diseases and industrial hygiene

Indian legal and institutional framework

Prevention, surveillance and measurement in practice

Key points

Formulas and examples

AQI sub-index Ip = [(IHi − ILo)/(BHi − BLo)] × (Cp − BLo) + ILo — Cp = pollutant concentration; BLo/BHi = breakpoint concentrations bracketing Cp; ILo/IHi = corresponding index values. Overall AQI = max(all sub-indices).
8-hour TWA = Σ(Ci × ti)/8 — Ci = concentration in interval i (mg/m³ or ppm), ti = duration in hours.
Dust/gas dose equivalence: mg/m³ = (ppm × molecular weight)/24.45 at 25 °C and 1 atm.
Permissible noise exposure (5 dB exchange rule, Indian practice): T = 8 / 2(L − 90)/5 hours — L = sound level in dB(A), T = allowed duration.
Noise dose D = Σ(Ci/Ti) × 100 % — Ci = actual hours at a level, Ti = permitted hours at that level; D > 100% exceeds the limit.
Combining noise sources: Ltotal = 10 log10 Σ 10Li/10 dB.
Accident frequency rate = (number of lost-time injuries × 1,000,000)/man-hours worked; Severity rate = (days lost × 1,000,000)/man-hours worked; Incidence rate = (injuries × 1,000)/average number of workers.
Attributable risk percent in the exposed = [(Ie − Iu)/Ie] × 100 — Ie, Iu = incidence in exposed and unexposed; Population attributable fraction = Pe(RR − 1)/[Pe(RR − 1) + 1], Pe = proportion exposed.
Chlorine demand = chlorine applied − free residual chlorine (mg/L); break-point chlorination is applied beyond the break-point to leave free residual.
Ventilation requirement Q = G/(C − C0) — Q = fresh air flow (m³/h), G = contaminant generation rate, C = target concentration, C0 = inlet concentration.

Example 1 — 8-hour TWA against a limit. A worker in a paint shop is exposed to toluene: 2 hours at 120 ppm, 3 hours at 60 ppm, 2 hours at 30 ppm and 1 hour at 0 ppm (break).

Example 2 — noise dose and permitted time. A press operator spends 4 hours at 95 dB(A) and 3 hours at 90 dB(A).

Example 3 — AQI from PM2.5. A monitoring station in Kanpur records a 24-hour average PM2.5 of 148 µg/m³. The Indian AQI breakpoints for PM2.5 are 0–30 (0–50), 31–60 (51–100), 61–90 (101–200), 91–120 (201–300), 121–250 (301–400).

Example 4 — accident frequency and severity. A textile mill employs 600 workers working 8 hours a day for 300 days; in one year there were 18 lost-time injuries and 540 man-days lost.

Example 5 — attributable risk in a silica-exposed cohort. Chest-X-ray-confirmed silicosis is found in 90 of 600 stone-crusher workers and in 6 of 600 matched unexposed men.

DiseaseAgent / industryKey clinical pointerPrevention
SilicosisFree silica; quarrying, stone crushing, foundry, agateUpper-zone nodular fibrosis, egg-shell hilar calcification, TB co-infectionWet drilling, LEV, enclosure, respirators, periodic X-ray and TB screening
AsbestosisAsbestos; sheets, brake linings, insulationBasal fibrosis, pleural plaques, mesothelioma decades laterSubstitution, strict control, ban on friable use, long-term surveillance
ByssinosisCotton, flax, hemp dustChest tightness on the first working day of the weekDust extraction, humidity control, spirometry monitoring
BagassosisMouldy sugarcane bagasseHypersensitivity pneumonitis 1–3 weeks after exposureKeep bagasse moisture >20%, spray with 2% propionic acid, masks
NIHLNoise >85–90 dB(A)4 kHz notch, bilateral sensorineural lossEnclosure, damping, earplugs/muffs, audiometry
Lead poisoningBattery, smelting, paint, printingAnaemia with punctate basophilia, colic, wrist dropSubstitution, LEV, hygiene, blood lead monitoring
Environmental healthOccupational health
PopulationWhole community, including children, elderly, sickWorking-age adults, usually screened fit
ExposureInvoluntary, lower concentration, 24 hoursOften higher concentration, 8 hours, known agents
StandardsNAAQS, IS 10500, Noise Rules — ambient limitsPEL/TWA schedules under the OSH&WC Code rules
RegulatorMoEFCC, CPCB/SPCB, CAQM, NGTLabour ministry, DGFASLI, state inspectorates, ESIC
CompensationCivil/tort, public liability insurance, NGT reliefStatutory employment-injury benefit under the Social Security Code

Where it is used

Interview questions

Common mistakes

Related topics

Waste management rules: plastic, e-waste and biomedical · Epidemiology: measures of association and study designs · Communicable disease control and vector-borne diseases · Health policy, legislation and regulation in India · Disaster management and emergency public health · Biostatistics and survey methods.

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